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Artigo de periodico
Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling (2014)
Protzek, André O. P.; Costa-Júnior, José M.; Rezende, Luiz F.; Santos, Gustavo J.; Araújo, Tiago Gomes; Vettorazzi, Jean F.; Ortis, Fernanda ; Carneiro, Everardo M.; Rafacho, Alex; Boschero, Antonio C.
Source: International Journal of Endocrinology. Unidade: ICB
Subjects: HISTOLOGIA, INSULINA (RESISTÊNCIA), CAMUNDONGOS, RATOS WISTAR
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ABNT
PROTZEK, André O. P. et al. Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling. International Journal of Endocrinology, v. 2014, p. 1-14, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/983453. Acesso em: 13 maio 2024.
APA
Protzek, A. O. P., Costa-Júnior, J. M., Rezende, L. F., Santos, G. J., Araújo, T. G., Vettorazzi, J. F., et al. (2014). Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling. International Journal of Endocrinology, 2014, 1-14. doi:10.1155/2014/983453
NLM
Protzek AOP, Costa-Júnior JM, Rezende LF, Santos GJ, Araújo TG, Vettorazzi JF, Ortis F, Carneiro EM, Rafacho A, Boschero AC. Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling [Internet]. International Journal of Endocrinology. 2014 ; 2014 1-14.[citado 2024 maio 13 ] Available from: https://doi.org/10.1155/2014/983453
Vancouver
Protzek AOP, Costa-Júnior JM, Rezende LF, Santos GJ, Araújo TG, Vettorazzi JF, Ortis F, Carneiro EM, Rafacho A, Boschero AC. Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling [Internet]. International Journal of Endocrinology. 2014 ; 2014 1-14.[citado 2024 maio 13 ] Available from: https://doi.org/10.1155/2014/983453
Artigo de periodico
Insulin release, peripheral insulin resistance and muscle function in protein malnutrition: a role of tricarboxylic acid cycle anaplerosis (2010)
Zoppi, Claudio C.; Silveira, Leonardo dos Reis; Oliveira, Camila A. M.; Boschero, Antonio C.; Curi, Rui; Carneiro, Everardo M.
Source: British Journal of Nutrition. Unidades: EEFERP, ICB
Assunto: FISIOLOGIA
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ABNT
ZOPPI, Claudio C. et al. Insulin release, peripheral insulin resistance and muscle function in protein malnutrition: a role of tricarboxylic acid cycle anaplerosis. British Journal of Nutrition, v. 103, n. 9, p. 1237-1250, 2010Tradução . . Disponível em: https://doi.org/10.1017/S0007114509993060. Acesso em: 13 maio 2024.
APA
Zoppi, C. C., Silveira, L. dos R., Oliveira, C. A. M., Boschero, A. C., Curi, R., & Carneiro, E. M. (2010). Insulin release, peripheral insulin resistance and muscle function in protein malnutrition: a role of tricarboxylic acid cycle anaplerosis. British Journal of Nutrition, 103( 9), 1237-1250. doi:10.1017/S0007114509993060
NLM
Zoppi CC, Silveira L dos R, Oliveira CAM, Boschero AC, Curi R, Carneiro EM. Insulin release, peripheral insulin resistance and muscle function in protein malnutrition: a role of tricarboxylic acid cycle anaplerosis [Internet]. British Journal of Nutrition. 2010 ; 103( 9): 1237-1250.[citado 2024 maio 13 ] Available from: https://doi.org/10.1017/S0007114509993060
Vancouver
Zoppi CC, Silveira L dos R, Oliveira CAM, Boschero AC, Curi R, Carneiro EM. Insulin release, peripheral insulin resistance and muscle function in protein malnutrition: a role of tricarboxylic acid cycle anaplerosis [Internet]. British Journal of Nutrition. 2010 ; 103( 9): 1237-1250.[citado 2024 maio 13 ] Available from: https://doi.org/10.1017/S0007114509993060
Artigo de periodico
Smad5 regulates Akt2 expression and insulin-induced glucose uptake in L6 myotubes (2010)
Anhê, Fernando Forato; Lellis-Santos, Camilo; Leite, Adriana R.; Hirabara, Sandro M.; Boschero, Antonio C.; Curi, Rui; Anhê, Gabriel Forato; Bordin, Silvana
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
ANHÊ, Fernando Forato et al. Smad5 regulates Akt2 expression and insulin-induced glucose uptake in L6 myotubes. Molecular and Cellular Endocrinology, v. 319, n. 1/2, p. 30-8, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2010.01.003. Acesso em: 13 maio 2024.
APA
Anhê, F. F., Lellis-Santos, C., Leite, A. R., Hirabara, S. M., Boschero, A. C., Curi, R., et al. (2010). Smad5 regulates Akt2 expression and insulin-induced glucose uptake in L6 myotubes. Molecular and Cellular Endocrinology, 319( 1/2), 30-8. doi:10.1016/j.mce.2010.01.003
NLM
Anhê FF, Lellis-Santos C, Leite AR, Hirabara SM, Boschero AC, Curi R, Anhê GF, Bordin S. Smad5 regulates Akt2 expression and insulin-induced glucose uptake in L6 myotubes [Internet]. Molecular and Cellular Endocrinology. 2010 ; 319( 1/2): 30-8.[citado 2024 maio 13 ] Available from: https://doi.org/10.1016/j.mce.2010.01.003
Vancouver
Anhê FF, Lellis-Santos C, Leite AR, Hirabara SM, Boschero AC, Curi R, Anhê GF, Bordin S. Smad5 regulates Akt2 expression and insulin-induced glucose uptake in L6 myotubes [Internet]. Molecular and Cellular Endocrinology. 2010 ; 319( 1/2): 30-8.[citado 2024 maio 13 ] Available from: https://doi.org/10.1016/j.mce.2010.01.003
Artigo de periodico
Augmentation of insulin secretion by leucine supplementation in malnourished rats: possible involvement of the phosphatidylinositol 3-phosphate kinase/mammalian target protein of rapamycin pathway (2010)
Filiputti, Eliane; Rafacho, Alex; Araújo, Eliana P.; Silveira, Leonardo R.; Trevisan, Amon; Batista, Thiago M.; Curi, Rui; Velloso, Lício A.; Quesada, Ivan; Boschero, Antonio C.; Carneiro, Everardo M.
Source: Metabolism: clinical and experimental. Unidades: EEFERP, ICB
Assunto: FISIOLOGIA
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ABNT
FILIPUTTI, Eliane et al. Augmentation of insulin secretion by leucine supplementation in malnourished rats: possible involvement of the phosphatidylinositol 3-phosphate kinase/mammalian target protein of rapamycin pathway. Metabolism: clinical and experimental, v. 59, n. 5, p. 635-44, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.metabol.2009.09.007. Acesso em: 13 maio 2024.
APA
Filiputti, E., Rafacho, A., Araújo, E. P., Silveira, L. R., Trevisan, A., Batista, T. M., et al. (2010). Augmentation of insulin secretion by leucine supplementation in malnourished rats: possible involvement of the phosphatidylinositol 3-phosphate kinase/mammalian target protein of rapamycin pathway. Metabolism: clinical and experimental, 59( 5), 635-44. doi:10.1016/j.metabol.2009.09.007
NLM
Filiputti E, Rafacho A, Araújo EP, Silveira LR, Trevisan A, Batista TM, Curi R, Velloso LA, Quesada I, Boschero AC, Carneiro EM. Augmentation of insulin secretion by leucine supplementation in malnourished rats: possible involvement of the phosphatidylinositol 3-phosphate kinase/mammalian target protein of rapamycin pathway [Internet]. Metabolism: clinical and experimental. 2010 ; 59( 5): 635-44.[citado 2024 maio 13 ] Available from: https://doi.org/10.1016/j.metabol.2009.09.007
Vancouver
Filiputti E, Rafacho A, Araújo EP, Silveira LR, Trevisan A, Batista TM, Curi R, Velloso LA, Quesada I, Boschero AC, Carneiro EM. Augmentation of insulin secretion by leucine supplementation in malnourished rats: possible involvement of the phosphatidylinositol 3-phosphate kinase/mammalian target protein of rapamycin pathway [Internet]. Metabolism: clinical and experimental. 2010 ; 59( 5): 635-44.[citado 2024 maio 13 ] Available from: https://doi.org/10.1016/j.metabol.2009.09.007
Artigo de periodico
Deletion of tumor necrosis factor-α receptor 1 (TNFR1) Protects against diet-induced obesity by means of Increased thermogenesis (2009)
Romanatto, Talita; Roman, Erika A.; Denis, Raphael G.; Solon, Carina; Milanski, Marciane; Moraes, Juliana C.; Bonfleur, Maria L.; Degasperi, Giovanna R.; Picardi, Paty K.; Hirabara, Sandro Massao; Boschero, Antonio C.; Velloso, Licio; Curi, Rui
Source: Journal of Biological Chemistry. Unidades: FZEA, ICB
Subjects: FISIOLOGIA, NECROSE
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ABNT
ROMANATTO, Talita et al. Deletion of tumor necrosis factor-α receptor 1 (TNFR1) Protects against diet-induced obesity by means of Increased thermogenesis. Journal of Biological Chemistry, v. 284, n. 52, p. 36213-36222, 2009Tradução . . Disponível em: https://doi.org/10.1074/jbc.a109.030874. Acesso em: 13 maio 2024.
APA
Romanatto, T., Roman, E. A., Denis, R. G., Solon, C., Milanski, M., Moraes, J. C., et al. (2009). Deletion of tumor necrosis factor-α receptor 1 (TNFR1) Protects against diet-induced obesity by means of Increased thermogenesis. Journal of Biological Chemistry, 284( 52), 36213-36222. doi:10.1074/jbc.a109.030874
NLM
Romanatto T, Roman EA, Denis RG, Solon C, Milanski M, Moraes JC, Bonfleur ML, Degasperi GR, Picardi PK, Hirabara SM, Boschero AC, Velloso L, Curi R. Deletion of tumor necrosis factor-α receptor 1 (TNFR1) Protects against diet-induced obesity by means of Increased thermogenesis [Internet]. Journal of Biological Chemistry. 2009 ; 284( 52): 36213-36222.[citado 2024 maio 13 ] Available from: https://doi.org/10.1074/jbc.a109.030874
Vancouver
Romanatto T, Roman EA, Denis RG, Solon C, Milanski M, Moraes JC, Bonfleur ML, Degasperi GR, Picardi PK, Hirabara SM, Boschero AC, Velloso L, Curi R. Deletion of tumor necrosis factor-α receptor 1 (TNFR1) Protects against diet-induced obesity by means of Increased thermogenesis [Internet]. Journal of Biological Chemistry. 2009 ; 284( 52): 36213-36222.[citado 2024 maio 13 ] Available from: https://doi.org/10.1074/jbc.a109.030874
Artigo de periodico
Islet neogenesis-associated protein signaling in neonatal pancreatic rat islets: involvement of the cholinergic pathway (2008)
Barbosa, Helena C.; Bordin, Silvana ; Anhê, Gabriel Forato; Persaud, Shanta J.; Bowe, James; Borelli, Maria I.; Gagliardino, Juan J.; Boschero, Antonio C.
Source: Journal of Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
BARBOSA, Helena C. et al. Islet neogenesis-associated protein signaling in neonatal pancreatic rat islets: involvement of the cholinergic pathway. Journal of Endocrinology, v. 199, n. 2, p. 299-306, 2008Tradução . . Disponível em: https://doi.org/10.1677/joe-08-0309. Acesso em: 13 maio 2024.
APA
Barbosa, H. C., Bordin, S., Anhê, G. F., Persaud, S. J., Bowe, J., Borelli, M. I., et al. (2008). Islet neogenesis-associated protein signaling in neonatal pancreatic rat islets: involvement of the cholinergic pathway. Journal of Endocrinology, 199( 2), 299-306. doi:10.1677/joe-08-0309
NLM
Barbosa HC, Bordin S, Anhê GF, Persaud SJ, Bowe J, Borelli MI, Gagliardino JJ, Boschero AC. Islet neogenesis-associated protein signaling in neonatal pancreatic rat islets: involvement of the cholinergic pathway [Internet]. Journal of Endocrinology. 2008 ; 199( 2): 299-306.[citado 2024 maio 13 ] Available from: https://doi.org/10.1677/joe-08-0309
Vancouver
Barbosa HC, Bordin S, Anhê GF, Persaud SJ, Bowe J, Borelli MI, Gagliardino JJ, Boschero AC. Islet neogenesis-associated protein signaling in neonatal pancreatic rat islets: involvement of the cholinergic pathway [Internet]. Journal of Endocrinology. 2008 ; 199( 2): 299-306.[citado 2024 maio 13 ] Available from: https://doi.org/10.1677/joe-08-0309
Artigo de periodico
Signal transducer and activator of transcription 3-regulated sarcoendoplasmic reticulum 'Ca POT. 2+'-ATPase 2 expression by prolactin and glucocorticoids is involved in the adaptation of insulin secretory response during the peripartum period (2007)
Anhê, Gabriel Forato; Nogueira, Tatiane C. A.; Nicoletti-Carvalho, José Edgar; Lellis-Santos, Camilo; Barbosa, Helena C.; Cipolla Neto, José; Bosqueiro, José R.; Boschero, Antonio C.; Bordin, Silvana
Source: Journal of Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
ANHÊ, Gabriel Forato et al. Signal transducer and activator of transcription 3-regulated sarcoendoplasmic reticulum 'Ca POT. 2+'-ATPase 2 expression by prolactin and glucocorticoids is involved in the adaptation of insulin secretory response during the peripartum period. Journal of Endocrinology, v. 195, n. 1, p. 17-27, 2007Tradução . . Acesso em: 13 maio 2024.
APA
Anhê, G. F., Nogueira, T. C. A., Nicoletti-Carvalho, J. E., Lellis-Santos, C., Barbosa, H. C., Cipolla Neto, J., et al. (2007). Signal transducer and activator of transcription 3-regulated sarcoendoplasmic reticulum 'Ca POT. 2+'-ATPase 2 expression by prolactin and glucocorticoids is involved in the adaptation of insulin secretory response during the peripartum period. Journal of Endocrinology, 195( 1), 17-27.
NLM
Anhê GF, Nogueira TCA, Nicoletti-Carvalho JE, Lellis-Santos C, Barbosa HC, Cipolla Neto J, Bosqueiro JR, Boschero AC, Bordin S. Signal transducer and activator of transcription 3-regulated sarcoendoplasmic reticulum 'Ca POT. 2+'-ATPase 2 expression by prolactin and glucocorticoids is involved in the adaptation of insulin secretory response during the peripartum period. Journal of Endocrinology. 2007 ; 195( 1): 17-27.[citado 2024 maio 13 ]
Vancouver
Anhê GF, Nogueira TCA, Nicoletti-Carvalho JE, Lellis-Santos C, Barbosa HC, Cipolla Neto J, Bosqueiro JR, Boschero AC, Bordin S. Signal transducer and activator of transcription 3-regulated sarcoendoplasmic reticulum 'Ca POT. 2+'-ATPase 2 expression by prolactin and glucocorticoids is involved in the adaptation of insulin secretory response during the peripartum period. Journal of Endocrinology. 2007 ; 195( 1): 17-27.[citado 2024 maio 13 ]
Artigo de periodico
Phosphoinositide-specific inositol polyphosphate 5- phosphatase IV inhibits inositide trisphosphate accumulation in hypothalamus and regulates food intake and body weight (2006)
Bertelli, Daniela F.; Araújo, Eliana P.; Cesquini, Maristela; Stoppa, Graziela R.; Gasparotto-Contessotto, Miriam; Toyama, Marcos H.; Felix, Jorge Vinicius Cestari; Carvalheira, José B.; Michelini, Lisete Compagno ; Chiavegatto, Silvana ; Boschero, Antonio C.; Saad, Mario J. A.; Lopes-Cendes, Iscia; Velloso, Lício Augusto
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
BERTELLI, Daniela F. et al. Phosphoinositide-specific inositol polyphosphate 5- phosphatase IV inhibits inositide trisphosphate accumulation in hypothalamus and regulates food intake and body weight. Endocrinology, v. 147, n. 11, p. 5385-5399, 2006Tradução . . Acesso em: 13 maio 2024.
APA
Bertelli, D. F., Araújo, E. P., Cesquini, M., Stoppa, G. R., Gasparotto-Contessotto, M., Toyama, M. H., et al. (2006). Phosphoinositide-specific inositol polyphosphate 5- phosphatase IV inhibits inositide trisphosphate accumulation in hypothalamus and regulates food intake and body weight. Endocrinology, 147( 11), 5385-5399.
NLM
Bertelli DF, Araújo EP, Cesquini M, Stoppa GR, Gasparotto-Contessotto M, Toyama MH, Felix JVC, Carvalheira JB, Michelini LC, Chiavegatto S, Boschero AC, Saad MJA, Lopes-Cendes I, Velloso LA. Phosphoinositide-specific inositol polyphosphate 5- phosphatase IV inhibits inositide trisphosphate accumulation in hypothalamus and regulates food intake and body weight. Endocrinology. 2006 ; 147( 11): 5385-5399.[citado 2024 maio 13 ]
Vancouver
Bertelli DF, Araújo EP, Cesquini M, Stoppa GR, Gasparotto-Contessotto M, Toyama MH, Felix JVC, Carvalheira JB, Michelini LC, Chiavegatto S, Boschero AC, Saad MJA, Lopes-Cendes I, Velloso LA. Phosphoinositide-specific inositol polyphosphate 5- phosphatase IV inhibits inositide trisphosphate accumulation in hypothalamus and regulates food intake and body weight. Endocrinology. 2006 ; 147( 11): 5385-5399.[citado 2024 maio 13 ]
Artigo de periodico
Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus (2005)
Souza, Cláudio T. de; Araujo, Eliana P.; Bordin, Silvana ; Ashimine, Rika; Zollner, Ricardo L.; Boschero, Antonio C.; Saad, Mário J. A.; Velloso, Lício Augusto
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
SOUZA, Cláudio T. de et al. Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus. Endocrinology, v. 146, n. 10, p. 4192-4199, 2005Tradução . . Acesso em: 13 maio 2024.
APA
Souza, C. T. de, Araujo, E. P., Bordin, S., Ashimine, R., Zollner, R. L., Boschero, A. C., et al. (2005). Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus. Endocrinology, 146( 10), 4192-4199.
NLM
Souza CT de, Araujo EP, Bordin S, Ashimine R, Zollner RL, Boschero AC, Saad MJA, Velloso LA. Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus. Endocrinology. 2005 ; 146( 10): 4192-4199.[citado 2024 maio 13 ]
Vancouver
Souza CT de, Araujo EP, Bordin S, Ashimine R, Zollner RL, Boschero AC, Saad MJA, Velloso LA. Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus. Endocrinology. 2005 ; 146( 10): 4192-4199.[citado 2024 maio 13 ]
Artigo de periodico
Participation of prolactin receptors and phosphatidylinositol 3-kinase and MAP kinase pathways in the increase in pancreatic islet mass and sensitivity to glucose during pregnancy (2004)
Amaral, Maria E. C.; Cunha, Daniel A.; Anhê, Gabriel Forato; Ueno, Mirian; Carneiro, Everardo M.; Velloso, Lício Augusto; Bordin, Silvana ; Boschero, Antonio C.
Source: Journal of Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
AMARAL, Maria E. C. et al. Participation of prolactin receptors and phosphatidylinositol 3-kinase and MAP kinase pathways in the increase in pancreatic islet mass and sensitivity to glucose during pregnancy. Journal of Endocrinology, v. 183, p. 469-476, 2004Tradução . . Disponível em: https://doi.org/10.1677/joe.1.05547. Acesso em: 13 maio 2024.
APA
Amaral, M. E. C., Cunha, D. A., Anhê, G. F., Ueno, M., Carneiro, E. M., Velloso, L. A., et al. (2004). Participation of prolactin receptors and phosphatidylinositol 3-kinase and MAP kinase pathways in the increase in pancreatic islet mass and sensitivity to glucose during pregnancy. Journal of Endocrinology, 183, 469-476. doi:10.1677/joe.1.05547
NLM
Amaral MEC, Cunha DA, Anhê GF, Ueno M, Carneiro EM, Velloso LA, Bordin S, Boschero AC. Participation of prolactin receptors and phosphatidylinositol 3-kinase and MAP kinase pathways in the increase in pancreatic islet mass and sensitivity to glucose during pregnancy [Internet]. Journal of Endocrinology. 2004 ; 183 469-476.[citado 2024 maio 13 ] Available from: https://doi.org/10.1677/joe.1.05547
Vancouver
Amaral MEC, Cunha DA, Anhê GF, Ueno M, Carneiro EM, Velloso LA, Bordin S, Boschero AC. Participation of prolactin receptors and phosphatidylinositol 3-kinase and MAP kinase pathways in the increase in pancreatic islet mass and sensitivity to glucose during pregnancy [Internet]. Journal of Endocrinology. 2004 ; 183 469-476.[citado 2024 maio 13 ] Available from: https://doi.org/10.1677/joe.1.05547
Artigo de periodico
Prolactin-modulated gene expression profiles in pancreatic islets from adult female rats (2004)
Bordin, Silvana ; Amaral, Maria E. C.; Anhê, Gabriel Forato; Delghingaro-Augusto, Viviane; Cunha, Daniel A.; Nicoletti-Carvalho, José E.; Boschero, Antonio C.
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
BORDIN, Silvana et al. Prolactin-modulated gene expression profiles in pancreatic islets from adult female rats. Molecular and Cellular Endocrinology, v. 220, p. 41-50, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2004.04.001. Acesso em: 13 maio 2024.
APA
Bordin, S., Amaral, M. E. C., Anhê, G. F., Delghingaro-Augusto, V., Cunha, D. A., Nicoletti-Carvalho, J. E., & Boschero, A. C. (2004). Prolactin-modulated gene expression profiles in pancreatic islets from adult female rats. Molecular and Cellular Endocrinology, 220, 41-50. doi:10.1016/j.mce.2004.04.001
NLM
Bordin S, Amaral MEC, Anhê GF, Delghingaro-Augusto V, Cunha DA, Nicoletti-Carvalho JE, Boschero AC. Prolactin-modulated gene expression profiles in pancreatic islets from adult female rats [Internet]. Molecular and Cellular Endocrinology. 2004 ; 220 41-50.[citado 2024 maio 13 ] Available from: https://doi.org/10.1016/j.mce.2004.04.001
Vancouver
Bordin S, Amaral MEC, Anhê GF, Delghingaro-Augusto V, Cunha DA, Nicoletti-Carvalho JE, Boschero AC. Prolactin-modulated gene expression profiles in pancreatic islets from adult female rats [Internet]. Molecular and Cellular Endocrinology. 2004 ; 220 41-50.[citado 2024 maio 13 ] Available from: https://doi.org/10.1016/j.mce.2004.04.001
Artigo de periodico
Blockade of IRS1 in isolated rat pancreatic islets improves glucose-induced insulin secretion (2002)
Araujo, Eliana P.; Amaral, Maria E C.; Souza, Claudio T.; Bordin, Silvana ; Ferreira, Fabiano; Saad, Mario J A.; Boschero, Antonio C.; Magalhães, Everardo C.; Velloso, Lício Augusto
Source: FEBS Letters. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
ARAUJO, Eliana P. et al. Blockade of IRS1 in isolated rat pancreatic islets improves glucose-induced insulin secretion. FEBS Letters, v. 531, p. 437-442, 2002Tradução . . Disponível em: https://doi.org/10.1016/s0014-5793(02)03580-9. Acesso em: 13 maio 2024.
APA
Araujo, E. P., Amaral, M. E. C., Souza, C. T., Bordin, S., Ferreira, F., Saad, M. J. A., et al. (2002). Blockade of IRS1 in isolated rat pancreatic islets improves glucose-induced insulin secretion. FEBS Letters, 531, 437-442. doi:10.1016/s0014-5793(02)03580-9
NLM
Araujo EP, Amaral MEC, Souza CT, Bordin S, Ferreira F, Saad MJA, Boschero AC, Magalhães EC, Velloso LA. Blockade of IRS1 in isolated rat pancreatic islets improves glucose-induced insulin secretion [Internet]. FEBS Letters. 2002 ;531 437-442.[citado 2024 maio 13 ] Available from: https://doi.org/10.1016/s0014-5793(02)03580-9
Vancouver
Araujo EP, Amaral MEC, Souza CT, Bordin S, Ferreira F, Saad MJA, Boschero AC, Magalhães EC, Velloso LA. Blockade of IRS1 in isolated rat pancreatic islets improves glucose-induced insulin secretion [Internet]. FEBS Letters. 2002 ;531 437-442.[citado 2024 maio 13 ] Available from: https://doi.org/10.1016/s0014-5793(02)03580-9

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